Evaluation of Successful percutaneous coronary intervention for right coronary artery chronic total occlusion on improvement of Right ventricular systolic function and reverse remodeling | ||||
| Al-Azhar International Medical Journal | ||||
| Volume 2023, Issue 1, January 2023 | ||||
| DOI: 10.58675/2682-339X.1916 | ||||
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| Authors | ||||
| Mostafa Attia Al Sawasany; Ahmad Mohammad Hassaan; Hazem Khaled Mustafa Abd El Hameed | ||||
| Department of Cardiology, Faculty of Medicine for boys, Al-Azhar University, Cairo, Egypt. | ||||
| Abstract | ||||
| Background: Lesions with TIMI grade 0 flow for over three months are chronic total occlusions. Chronic coronary occlusions cause ventricular arrhythmias and poor outcomes. CTO difficulties and failure rates were increased even among seasoned PCI operators. 38–50% of CTO patients had RCAs. Right ventricular echocardiography was disregarded in the past. After depending largely on qualitative assessment in past, American Society of Echocardiography & European Association of cardiovascular imaging now offer many quantitative methods for assessing the RV. Aim: To correlate the relationship between Successful percutaneous coronary intervention for right coronary artery chronic total occlusion & improvement of Right ventricular functions. Subjects & techniques: research contained 60 studied cases who had undergone coronary angiography at National Heart Institute, Giza, Egypt. Results: Hypertension was the most frequent risk factor found in most patients. Stress test revealed that all studied cases had viable RCA territory. Left main coronary artery was free in all cases & proximal segment of left anterior descending artery had lesion in some cases. Successful PCI was achieved in most studied cases. There was significant elevation in S` and TAPSE after intervention compared to results before intervention. Conclusion: RV function improves after percutaneous coronary intervention of a chronically and totally occluded artery. Successful CTO PCI improves RV and LV ejection fraction & decreases LV end systolic volume, suggesting favourable influence on LV and RV systolic function and remodeling. | ||||
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