Serum prolactin level in a patient with NSTE - ACS | ||||
Minia Journal of Medical Research | ||||
Volume 30, Issue 2, April 2019, Page 46-48 PDF (120.71 K) | ||||
Document Type: Original Article | ||||
DOI: 10.21608/mjmr.2022.221982 | ||||
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Authors | ||||
Amr S. Amin1; Lamiaa H. Abd Elhuq2; Sayed Sh. Mahmoud1; Taha A. Abd Elhameed3 | ||||
1Department of Cardiology, Faculty of Medicine - Minia University, | ||||
2Department of Clinical Pathology, Faculty of Medicine - Minia University | ||||
3* Department of Cardiology, Faculty of Medicine - Minia University | ||||
Abstract | ||||
Introduction: Acute coronary syndrome was defined as the cluster of symptoms arising due to the rapid drop of blood flow to the heart because of coronary artery obstruction. Aim of the work: The aim of this study is to determine. Would the level of prolactin detect early, diagnosis of NSTE – ACS??, Would the level of prolactin predict prognosis of NSTE - ACS??, Would the level of prolactin correlate with grace risk score??. Patients and Methods: Our study included 80 patients (Pts.) diagnosed as NSTE-ACS (NSTEMI and Unstable angina). The Pts were presented within 3hs from the onset of chest pain to our Coronary Care Unit of EL- Minia University hospital in the period from July 2016 to April 2017. Results: This study included 80 patients (Pts) who presented with NSTE-ACS within 3 hs from the onset of chest pain, whether un-stable angina or NSTEMI. The Pts were presented to our Coronary Care Unit of Minia University hospital in the period from July 2016 to April 2017. Discussion: Hyper-prolactinemia may reflect neuroendocrine stress reaction to ACS, which induces acute endothelial dysfunction, insulin resistance, and Induction of vascular immune reactions thus, prolonged hyper-prolactinemia lead to arteriosclerosis, augmentation of arterial stiffness, and hypertension. (Medic-Stojanoska et al., 2015). Summary: Our study included 80 consecutive Pts, [51 males (63.8%), 29 females (36.2%), with a mean age ± SD of 57.42 ± 10.3 years, diagnosed as NSTE-ACS at CCU of EL-Minia University Hospital in the period from July 2016 to April 2017. | ||||
Keywords | ||||
VEGF-B : Vascular Endothelial Growth Factor -B; VIP: Vasoactive Intestinal Polypeptide; VSMC: Vascular Smooth Muscle Cells | ||||
Supplementary Files
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